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Lipopolysaccharide-Induced Microglial Activation and Neuroprotection  against Experimental Brain Injury Is Independent of Hematogenous TLR4 |  Journal of Neuroscience
Lipopolysaccharide-Induced Microglial Activation and Neuroprotection against Experimental Brain Injury Is Independent of Hematogenous TLR4 | Journal of Neuroscience

Exploring New Inflammatory Biomarkers and Pathways during LPS-Induced M1  Polarization
Exploring New Inflammatory Biomarkers and Pathways during LPS-Induced M1 Polarization

Hydrogen Sulfide Reverses LPS-Induced Behavioral Deficits by Suppressing  Microglial Activation and Promoting M2 Polarization | SpringerLink
Hydrogen Sulfide Reverses LPS-Induced Behavioral Deficits by Suppressing Microglial Activation and Promoting M2 Polarization | SpringerLink

Microglia activation visualization via fluorescence lifetime imaging  microscopy of intrinsically fluorescent metabolic cofactors
Microglia activation visualization via fluorescence lifetime imaging microscopy of intrinsically fluorescent metabolic cofactors

Microglia-triggered hyperexcitability in the cerebellum depresses animal  behaviors | bioRxiv
Microglia-triggered hyperexcitability in the cerebellum depresses animal behaviors | bioRxiv

Chronic Intermittent Ethanol and Lipopolysaccharide Exposure Differentially  Alter Iba-1-Derived Microglia Morphology in the Prelimbic Cortex and  Nucleus Accumbens Core | bioRxiv
Chronic Intermittent Ethanol and Lipopolysaccharide Exposure Differentially Alter Iba-1-Derived Microglia Morphology in the Prelimbic Cortex and Nucleus Accumbens Core | bioRxiv

PLOS ONE: Production of proinflammatory mediators in activated microglia is  synergistically regulated by Notch-1, glycogen synthase kinase (GSK-3β) and  NF-κB/p65 signalling
PLOS ONE: Production of proinflammatory mediators in activated microglia is synergistically regulated by Notch-1, glycogen synthase kinase (GSK-3β) and NF-κB/p65 signalling

Figure 2 | Inflammatory Animal Model for Parkinson's Disease: The  Intranigral Injection of LPS Induced the Inflammatory Process along with  the Selective Degeneration of Nigrostriatal Dopaminergic Neurons
Figure 2 | Inflammatory Animal Model for Parkinson's Disease: The Intranigral Injection of LPS Induced the Inflammatory Process along with the Selective Degeneration of Nigrostriatal Dopaminergic Neurons

Peroxynitrite generated by inducible nitric oxide synthase and NADPH  oxidase mediates microglial toxicity to oligodendrocytes | PNAS
Peroxynitrite generated by inducible nitric oxide synthase and NADPH oxidase mediates microglial toxicity to oligodendrocytes | PNAS

Lipopolysaccharide induces neuroinflammation in microglia by activating the  MTOR pathway and downregulating Vps34 to inhibit autophagosome formation |  Journal of Neuroinflammation | Full Text
Lipopolysaccharide induces neuroinflammation in microglia by activating the MTOR pathway and downregulating Vps34 to inhibit autophagosome formation | Journal of Neuroinflammation | Full Text

Sesamin suppresses LPS-induced microglial activation via regulation of TLR4  expression - ScienceDirect
Sesamin suppresses LPS-induced microglial activation via regulation of TLR4 expression - ScienceDirect

Acetate Suppresses Lipopolysaccharide-stimulated Nitric Oxide Production in  Primary Rat Microglia but not in BV-2 Microglia Cells | Bentham Science
Acetate Suppresses Lipopolysaccharide-stimulated Nitric Oxide Production in Primary Rat Microglia but not in BV-2 Microglia Cells | Bentham Science

A unique hybrid characteristic having both pro- and anti-inflammatory  phenotype transformed by repetitive low-dose lipopolysaccharide in C8-B4  microglia | Scientific Reports
A unique hybrid characteristic having both pro- and anti-inflammatory phenotype transformed by repetitive low-dose lipopolysaccharide in C8-B4 microglia | Scientific Reports

TLR4-activated microglia require IFN-γ to induce severe neuronal  dysfunction and death in situ | PNAS
TLR4-activated microglia require IFN-γ to induce severe neuronal dysfunction and death in situ | PNAS

Morphological characteristics of activated microglia following systemic...  | Download Scientific Diagram
Morphological characteristics of activated microglia following systemic... | Download Scientific Diagram

Antioxidants | Free Full-Text | Hydroxytyrosol Decreases LPS- and  α-Synuclein-Induced Microglial Activation In Vitro
Antioxidants | Free Full-Text | Hydroxytyrosol Decreases LPS- and α-Synuclein-Induced Microglial Activation In Vitro

Systemic inflammation impairs microglial Aβ clearance through NLRP3  inflammasome | The EMBO Journal
Systemic inflammation impairs microglial Aβ clearance through NLRP3 inflammasome | The EMBO Journal

NADPH Oxidase Mediates Lipopolysaccharide-induced Neurotoxicity and  Proinflammatory Gene Expression in Activated Microglia* - Journal of  Biological Chemistry
NADPH Oxidase Mediates Lipopolysaccharide-induced Neurotoxicity and Proinflammatory Gene Expression in Activated Microglia* - Journal of Biological Chemistry

Single‐cell transcriptomics reveals distinct inflammation‐induced microglia  signatures | EMBO reports
Single‐cell transcriptomics reveals distinct inflammation‐induced microglia signatures | EMBO reports

Lipopolysaccharide induces neuroinflammation in microglia by activating the  MTOR pathway and downregulating Vps34 to inhibit autophagosome formation |  Journal of Neuroinflammation | Full Text
Lipopolysaccharide induces neuroinflammation in microglia by activating the MTOR pathway and downregulating Vps34 to inhibit autophagosome formation | Journal of Neuroinflammation | Full Text

Frontiers | Microglia Responses to Pro-inflammatory Stimuli (LPS,  IFNγ+TNFα) and Reprogramming by Resolving Cytokines (IL-4, IL-10) |  Cellular Neuroscience
Frontiers | Microglia Responses to Pro-inflammatory Stimuli (LPS, IFNγ+TNFα) and Reprogramming by Resolving Cytokines (IL-4, IL-10) | Cellular Neuroscience

Molecules | Free Full-Text | Antineuroinflammatory Activities and  Neurotoxicological Assessment of Curcumin Loaded Solid Lipid Nanoparticles  on LPS-Stimulated BV-2 Microglia Cell Models
Molecules | Free Full-Text | Antineuroinflammatory Activities and Neurotoxicological Assessment of Curcumin Loaded Solid Lipid Nanoparticles on LPS-Stimulated BV-2 Microglia Cell Models

Neurodegeneration by Activation of the Microglial Complement–Phagosome  Pathway | Journal of Neuroscience
Neurodegeneration by Activation of the Microglial Complement–Phagosome Pathway | Journal of Neuroscience

Umbilical cord-derived mesenchymal stromal cells immunomodulate and restore  actin dynamics and phagocytosis of LPS-activated microglia via PI3K/Akt/Rho  GTPase pathway | Cell Death Discovery
Umbilical cord-derived mesenchymal stromal cells immunomodulate and restore actin dynamics and phagocytosis of LPS-activated microglia via PI3K/Akt/Rho GTPase pathway | Cell Death Discovery